#SocialBehavior

A small incident in a quiet neighborhood—where a three-year-old named Manny told his playmate Albert the real facts about where babies come from, after learning it from his own parents—may seem trivial at first glance. But Albert’s parents, who had told him the traditional “stork story,” were furious, leading to a rift between the families and a ban on further playdates. This slice-of-life story, first published in Slate’s advice column on April 23, 2025, has struck a chord with parents around the world and spurred fresh conversations about what happens when children repeat what they learn at home with their peers—a dynamic that carries both social and developmental significance (Slate).

A groundbreaking international study has pinpointed a specific region in the brain responsible for deciding how generous we are with friends versus strangers. Researchers from Germany and South Africa have discovered that damage to the basolateral amygdala (BLA) sharply reduces our willingness to share with anyone outside our closest social circle—while generosity toward close friends stays intact. The findings, published recently in the Proceedings of the National Academy of Sciences, offer intriguing new insights into the biological roots of kindness and selfishness, and may have implications for understanding social disorders.

Recent research led by MIT and Harvard Medical School has uncovered the intriguing role that the immune molecule interleukin-17 (IL-17) plays in shaping social behavior and anxiety by acting directly on specific brain regions. This groundbreaking study reveals that IL-17 serves dual functions: enhancing sociability by reducing neuron excitability in the brain’s cortex and triggering anxiety by increasing excitability in the amygdala. These findings suggest IL-17 acts like a neuromodulator, drawing a fascinating link between immune system activity and behavioral outcomes, with potential implications for the treatment of conditions like autism and depression (Neuroscience News).